[1] 龙晓婷,刘俊磊,郭洪,等.伪狂犬病毒在潜伏感染猪体内的组织分布[J].畜牧兽医学报,2008,39(5):645-651. LONG X T,LIU J L,GUO H,et al.Distribution of pseudorabies virus in latently infected pigs[J].Acta Veterinaria et Zootechnica Sinica,2008,39(5):645-651.(in Chinese)
[2] 青宁生.动物病毒学家——殷震[J].微生物学报,2018,58(5):955-956. QING N S.An animal virologist——Yin Z[J].Acta Microbiologica Sinica,2018,58(5):955-956.(in Chinese)
[3] GUZ Q,HOUC C,SUN H F,et al.Emergence of highly virulent pseudorabies virus in Southern China[J].Canadian Journal of Veterinary Research,2015,79(3):221-228.
[4] WU R,BAI C Y,SUN J Z,et al.Emergence of virulent pseudorabies virus infection in Northern China[J].Journal of Veterinary Science,2013,14(3):363-365.
[5] ZHAO G N,JIANG D S,LI H.Interferon regulatory factors:At the crossroads of immunity,metabolism,and disease[J].Biochimica et Biophysica Acta,2015,1852(2):365-378.
[6] JUANG Y T,LOWTHER W,KELLUM M,et al.Primary activation of interferon A and interferon B gene transcription by interferon regulatory factor 3[J].Proceedings of the National Academy of Sciences,1998,95(17):9837-9842.
[7] BELLINGHAM J,GREGORY-EVANS K,GREGORY-EVANS C Y,et al.Mapping of human interferon regulatory factor 3(IRF3) to chromosome 19q13.3-13.4 by an intragenic polymorphic marker[J].Annals of Human Genetics,1998,62(Pt 3):231-234.
[8] TANIGUCHI T,OGASAWARA K,TAKAOKA A,et al.IRF family of transcription factors as regulators of host defense[J].Annual Review of Immunology,2001,19(1):623-655.
[9] OZATO K,TAILOR P,KUBOTA T.The interferon regulatory factor family in host defense:Mechanism of action[J].Journal of Biological Chemistry,2007,282(28):20065-20069.
[10] LIN R,HEYLBROECK C,PITHA P M,et al.Virus-dependent phosphorylation of the IRF-3 transcription factor regulates nuclear translocation,transactivation potential,and proteasome-mediated degradation[J].Molecular and Cellular Biology,1998,18(5):2986-2996.
[11] 李帅,赵一霏,沈国顺.一例猪伪狂犬病的诊断与防控建议[J].黑龙江畜牧兽医,2018,22:122-124. LI S,ZHAO Y Z,SHEN G S.A diagnosis and prevention and control of a case of pseudorabies in pigs[J]. Heilongjiang Animal Science and Veterinary Medicine,2018,22:122-124.(in Chinese)
[12] SAITO T,GALE M JR.Principles of intracellular viral recognition[J].Current Opinion Immunology,2007,19(1):17-23.
[13] WANG H,YANG H,SHIVALILA C S,et al.One-step generation of mice carrying mutations in multiple genes by CRISPR/Cas-mediated genome engineering[J].Cell,2013,153(4):910-918.
[14] KORKMAZ G,LOPES R,UGALDE A P,et al.Functional genetic screens for enhancer elements in the human genome using CRISPR-Cas9[J].Nature Biotechnology,2016,34(2):192-198.
[15] MAZ A,PERETZ M,AHARONY A,et al.Defining essential genes for human pluripotent stem cells by CRISPR-Cas9 screening in haploid cells[J]. Nature Cell Biology,2018,20(5):610-619.
[16] KLANN T S,BLACK J B,CHELLAPPAN M,et al.CRISPR-Cas9 epigenome editing enables high-throughput screening for functional regulatory elements in the human genome[J].Nature Biotechnology,2017,35(6):561-568.
[17] NELSON C A,FREMONT D H,VIRGIN H W,et al.Discovery of a proteinaceous cellular receptor for a norovirus[J].Science,2016,353(6302):933-936.
[18] HAGA K,FUJIMOTO A,TAKAI-TODAKA R,et al.Functional receptor molecules CD300lf and CD300ld within the CD300 family enable murine noroviruses to infect cells[J].Proceedings of the National Academy of Sciences of the United States of America,2016,113(41):E6248-E6255.
[19] VIRREIRA WINTER S,ZYCHLINSKY A,BARDOEL B W.Genome-wide CRISPR screen reveals novel host factors required for Staphylococcus aureus α-hemolysin-mediated toxicity[J].Scientific Reports,2016,6:242-244.
[20] ZHANG R,MINER J J,GORMAN M J,et al.A CRISPR screen defines a signal peptide processing pathway required by flaviviruses[J].Nature,2016,535(7610):164-168.
[21] MA H,DANG Y,WU Y,et al.A CRISPR-based screen identifies genes essential for West-Nile-virus-induced cell death[J].Cell Reports,2015,12(4):673-683.
[22] MARCEAU C D,PUSCHNIK A S,MAJZOUB K,et al.Genetic dissection of Flaviviridae host factors through genome-scale CRISPR screens[J].Nature,2016,535(7610):159-163.
[23] SIDIK S M,HUET D,GANESAN S M A,et al.A genome-wide CRISPR screen in toxoplasma identifies essential apicomplexan genes[J].Cell,2016,166(6):1423-1435.
[24] MODELL J W,JIANG W,MARRAFFINI L A.CRISPR-Cas systems exploit viral DNA injection to establish and maintain adaptive immunity[J].Nature,2017,544(7648):101-104.
[25] MOCH C,FROMANT M,BLANQUET S,et al.DNA binding specificities of Escherichia coli Cas1-Cas2 integrase drive its recruitment at the CRISPR locus[J].Nucleic Acids Research,2017,45(5):2714-2723.
[26] WRIGHT A V,LIU J J,KNOTT G J,et al.Structures of the CRISPR genome integration complex[J]. Science,2017,357(6356):1113-1118.
[27] SHALEM O,SANJANA N E,HARTENIAN E,et al.Genome-scale CRISPR-Cas9 knockout screening in human cells[J].Science,2014,343(6166):84-87.
[28] SHALEM O,SANJANA N E,ZHANG F.High-throughput functional genomics using CRISPR-Cas9[J].Nature Reviews Genetics,2015,16(5):299-311.
[29] MILLER L C,ZANELLA E L,WATERS W R,et al.Cytokine protein expression levels in tracheobronchial lymph node homogenates of pigs infected with pseudorabies virus[J].Clinical Vaccine Zmmanol,2010,17(5):728-734.
[30] VAN DER SANDEN S M,WU W,DYBDAHL-SISSOKO N,et al.Engineering enhanced vaccine cell lines to eradicate vaccine-preventable diseases:The polio end game[J].Journal of Virology,2015,90(4):1694-1704.
[31] BIKARD D,EULER C W,JIANG W,et al.Exploiting CRISPR-Cas nucleases to produce sequence-specific antimicrobials[J].Nature Biotechnology,2014,32(11):1146-1150. |